compleses re subgingival plaque. .1 Alin Periodontal 1998: 25: 134-144. Munksgaard. 1998, tat complex related strikingly to clinical measures of periodontal disease particu- larly pocket depth In deeper pockets, the species co-ex
22 May 2018 Subgingival plaque samples were collected from the first molars by means of a Meanwhile, the clinical diagnoses of periodontal disease and renal was higher than 95%, suggesting that the sequencing was deep enough.
79 ba in deep periodontal pockets subgingival anaerobes in deep periodon plaque removed. Group. 3. Subgingival and supragingival deposits removed. Group.
van Winkelhoff AJ(1), van der Velden U, de Graaff J. Author information: (1)Department of Oral Microbiology, Vrije Universiteit, Amsterdam, The Netherlands. 1. Shikai Tenbo. 1984 Aug;64(2):209-19. [A new method for the treatment of deep periodontal pockets. A new method for the control of subgingival plaque].
In the presence of supragingival plaque (Group A), however, a subgingival microbiota containing large numbers of spirochetes and motile rods was soon (4–8 weeks) reestablished. A small number of sites with deep pockets ( 8 mm) was not substantially reduced in depth following subgingival instrumentation.
The effect of supragingival plaque control on the subgingival microflora. Kho P, Smales FC, Hardie JM. The effect of plaque control on the apical microflora of deep periodontal pockets was studied.
Immediate effect of instrumentation on the subgingival microflora in deep inflamed pockets under strict plaque control. Rhemrev GE(1), Timmerman MF, Veldkamp I, Van Winkelhoff AJ, Van der Velden U. Author information: (1)Department of Periodontology, Academic Center for Dentistry Amsterdam (ACTA), Amsterdam, The Netherlands.
Human blood Human foot, deep infection, yr-old woman. Human Human subgingival plaque, chronic periodontitis. Prevotella outgrowth of low abundance disease-associated species from biofilms taken from healthy sites and subjects by mimicking a disease-promoting environment. Five days after intravenous immunoglobulin therapy, his deep sensory impairment in media mimicking the nutritional status of the inflamed periodontal pocket. Human periodontal pocket.
[A new method for the treatment of deep periodontal pockets. A new method for the control of subgingival plaque]. Immediate effect of instrumentation on the subgingival microflora in deep inflamed pockets under strict plaque control. Rhemrev GE(1), Timmerman MF, Veldkamp I, Van Winkelhoff AJ, Van der Velden U. Author information: (1)Department of Periodontology, Academic Center for Dentistry Amsterdam (ACTA), Amsterdam, The Netherlands. In the normal periodontium, the alveolar crest lies Subgingival plaque in deep periodontal pockets consists primarily of When placed into sound dentin, a self-threading pin will The microorganisms that initially colonize the tooth surface are associated with the genus The pulpal floor of an occlusal amalgam preparation on a mandibular first
Twenty‐one patients, with periodontal pockets 4 mm or deeper, underwent one session of scaling, root planing, and oral hygiene instruction (Bass method but without emphasis on interdental cleaning). Eight patients performed once daily subgingival irrigation with 0.2% chlorhexidine gluconate solution for 28 days.
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Address requests for reprints to Dr. Smulow. The effect of mechanical supragingival plaque control on the composition of the subgingival microflora in untreated 4‐6 mm deep periodontal pockets was investigated. 13 subjects with chronic periodontitis were recruited for the study. Periodontally‐diseased sites were subjected to professional plaque control 3 × weekly for a period of 3 weeks.
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Lindhe J, Hamp SE, Löe H. Plaque induced periodontal disease in beagle dogs. A 4-year clinical, erhållit professionell rengöring av tänderna, supragingival scaling och instruktion i tissue regeneration of deep periodontal pockets.
Vinholis et al. effect of subgingival irrigation with a 1% chlorhexidine collagen gel in periodontal pockets as an adjunct to The relationship of plaque is quite clear that "marginal" plaque and subgingival plaque are directly responsible for the initiation and progression of periodontal diseases. It is probable that Supragingival plaque strongly influences the growth, accumulation, and pathogenic potential of subgingival plaque, especially in the early stages of gingivitis and periodontitis. Although other factors, such as contaminated food or water (Slots el al.